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Mice lacking Nrf2 , a key transcriptional modulator of GCLC, start developing opacities at nine months and develop various age-related cataracts including advanced cortical, posterior subcapsular, anterior subcapsular, and nuclear cataracts (see below) [16]

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These models reveal differential effects of myelin in action potential propagation and axonal degeneration, bringing oligodendrocytes to the forefront of essential players in neuronal metabolic support