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Key mechanisms observed in pre-clinical research include: ERRα, ERRβ, and ERRγ activation — targeting the primary regulators of mitochondrial and metabolic gene expression, with highest potency at ERRα (EC₅₀ = 98 nM) PGC-1α upregulation — stimulating the so-called “exercise gene” that drives mitochondrial biogenesis Increased mitochondrial density — promoting the creation of new mitochondria within skeletal muscle cells, boosting cellular energy capacity Enhanced fatty acid oxidation — shifting cellular metabolism toward fat as a primary fuel source Improved oxidative metabolism — promoting efficient, oxygen-based energy production in muscle tissue Type IIa oxidative muscle fibre increases — promoting the development of endurance-associated fibre types in skeletal muscle models What Does SLU-PP-332 Do in Research