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Interestingly, while the role of GSTs has classically been associated to the modification of small molecules and the role of glutaredoxins to the deglutathionylation of proteins, it has recently been shown that human GSTO1-1 can deglutathionylate β-actin, which should prompt us to consider proteins as GST substrates (Menon and Board, 2013)

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Patients were randomized (2:1) to one of the following treatment arms: Treatment with Pemetrexed for Injection continued until RECIST v1.1 (modified to follow a maximum of 10 target lesions and a maximum of 5 target lesions per organ)-defined progression of disease as determined by the investigator or unacceptable toxicity