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Identification of two serine residues involved in agonist activation of the β-adrenergic receptor
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201 The contrasting effect of untargeted versus mitochondria-targeted antioxidants on tumorigenesis, although not generalisable to all tumour models, further corroborates the concept that tumour progression requires the generation of mitochondrial ROS and the limitation of cytosolic ROS, to maximise mitogenic signalling and avoid oxidative damage
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