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Increased resistance of AML cells to oxidative stress plays an important part in disease initiation and persistence and this characteristic has previously been attributed to increased NRF2 activity [8]

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Boxplots comparing protein expression of MTA1 between primitive and committed patients in both BeatAML-proteomics (n of primitive = 74 patients, n of committed = 74 patients) and CPTAC AML cohort (n of primitive = 75 patients, n of committed = 75 patients)